Triple-positive breast cancer, is it useful for women to take hormonal drugs?
It was useful for women with triple positive breast cancer to take hormonal drugs. Triple-positive breast cancer meant that the three indicators of the estrogen receptor, the hormone receptor, and the human embryonic growth factor receptor 2 were all positive. Because the estrogen receptor and the hormone receptor were positive, they could choose to use hormone therapy. For example, tamoxifen could be considered for hormonal therapy in pre-menstrual patients, and drugs such as alatrozuo, letrozuo, and eximestane could be considered for post-menstrual patients. The hormone therapy could regulate the level of hormones in the patient's body and suppress the growth of tumor cells. In addition, in different stages of treatment for triple positive breast cancer (such as auxiliary therapy, neo-auxiliary therapy, late-stage treatment, etc.), the combination of hormone therapy and other treatments (such as targeted therapy, etc.) could improve the treatment effect. Read more exciting novels for free
What are the side effects of oral hormonal drugs for breast cancer?
The following side effects may occur in breast cancer patients who take oral hormonal drugs: 1. Menopacus-like symptoms, such as hot flashes, night sweats, dry vaginas, itching, etc., may also appear anxiety, insomnia and other psychological symptoms, which is a direct manifestation of the decrease in the level of estrogens in the body caused by drugs. 2. Hot flashes, joint pain, bone loss, cardiovascular disease, etc. may occur. 3. For patients who took tamoxifen, there was a possibility of the development of Endometrial Cancer, so they needed to undergo regular gynecology examinations. 4. Taking an Aromatase Inhibition may cause bone thinning. 5. Luteinizing Hormone-Releasing Hormone-Anomalies may cause adverse reactions such as hot flashes, decreased libido, and mood changes. They may also have adverse reactions to bone thinning and the cardiovascular system. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
Is it useful to take targeted drugs for esophagus cancer metastasizing?
At present, a variety of targeted drugs were available for the treatment of esophagus cancer, including new small molecules multi-target Tyrosin Kinase Inhibition, drugs that targeted HEL2, cetuxed that targeted the egFr target, and bevacizumb that targeted the veg-like growth factor target, etc. There was also a combination of PD1 - 1 inhibition and targeted HEL2 for the treatment of cancer at the gastric-esophagus junction. Chinese studies have shown that as a new small-molecular multi-target Tyrosinase Kinase Inhibition, anlotinib and apitini have a certain effect in the treatment of esophagus cancer. The combination therapy of trastuzumab-targeted to HEL2 and PD1 inhibition drugs (such as pembrolizumab) combined with chemotherapy can be used as the first-line treatment for cancer of the gastroesophagus junction, advanced gastric cancer with HEL2 positive, and esophagus cancer. In the second-line treatment of China guidelines for the treatment of esophagus cancer, level II recommendations were given to anlotinib (Class 2A) and apitini (Class 3). However, there were no phase III clinical trials to prove that molecular targeted drugs could prolong the survival of patients with esophagus cancer. The effect of targeted therapy needed further study. In general, molecular targeted therapy had a certain clinical value for the treatment of patients with esophagus cancer, including those with metastasizing, but it still needed further in-depth research. These drugs must be used under the guidance of a professional doctor. It is not recommended for patients to buy and take them themselves to avoid serious adverse reactions. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
What are the drugs for early chemotherapy of breast cancer?
The liver-protecting drugs used for early chemotherapy of breast cancer included liver-protecting tablets, Glutathion tablets, Compound Ammine Glycerate injection, Diammine Glycerate Enteric capsules, Dicyclol tablets, and Yishanfu Polyene Phospholipidoline capsules. These drugs needed to be used under the guidance of a doctor. For example, liver-protecting tablets could protect the liver; Glutathione-based tablets could repair stem cells and detoxify; and compound licorice injection could protect the liver and improve the pain symptoms caused by liver damage. The bicyclol tablets had a good effect on reducing the index of Gu and C, but the effect on the index of Gu and Cao was not ideal. The Yishanfu Duoene Phospholidylcholin capsules did not improve the high index of transferment in some patients. Regarding the kidney protection drugs for early chemotherapy of breast cancer, the reference materials did not explicitly mention the relevant drugs. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
There are several types of breast cancer targeted drugs
There were mainly the following types of breast cancer targeted drugs: 1. Anti-neoplastic targeted drugs: There are intravenous preparations and oral drugs, but the effect in the treatment of breast cancer is not ideal. For example, bevacidol (Avastin) will be considered in some cases. 2. Anti-Her- 2 targeted drugs: targeted at HER - 2 positive patients, it is the most important targeted drug for breast cancer, mainly including trastuzumab (Herceptin), pertubizab, dm- 1, lapatini, etc. The prerequisite for use was that the patient was positive for HER - 2 (+++ in the immune tissue test). If the immune tissue test was ++, the patient would need to undergo a further FISH test. The patient could only be used if the FISH test was positive. If the immune tissue test was + or-, the patient could not be used. 3. A targeted drug for patients with fetal mutations in the Brca lineage: The main drug is olaparib, which is used to treat patients with metastasizing breast cancer who have harmful or suspected harmful mutations in the Brca lineage and who are negative for the human embryonic growth factor receptor 2 (HEL2). 4. Immune therapy (in a sense, it was also a targeted drug): Anti-PD- 1/PD-L1 Monoclone Antibodies, which essentially relieved immune suppression and activated immune cells to kill cancer cells. 5. mTO inhibition drugs, such as everolimus and sirolimus, can inhibit the activation of the mTO signaling pathway, thereby suppressing the growth and metabolism of tumor cells. 6. Other targeted therapy drugs, such as Parp inhibition drugs, CD 4/6 inhibition drugs, etc. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
What are the drugs for the early stage of breast cancer?
The drugs for early breast cancer include the following categories: 1. Hormonal receptor regulator, such as tamoxifen, toremifen, can be used in breast cancer patients with hormone receptor-positive before and after menstruation. 2. Aromatase inhibition drugs, such as letrozuo, anastrozuo, and eximestane, were mainly used in post-menstrual patients. They could also be used in combination with belly acupuncture before menstruation. 3. Ovary function suppressors (commonly known as belly needles): Representative drugs include goserelin and leuprorelin, which are suitable for breast cancer patients with premenstrual hormone receptor-positive. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
What are the side effects of breast cancer chemotherapy drugs?
The side effects of breast cancer chemotherapy drugs were as follows: 1. Bone marrow suppression: - White blood cell reduction was more common. For example, white blood cell reduction was more common after the use of cyclosporine, and the lowest value was 1 - 2 weeks after the use of cyclosporine. Most of them recovered after 2 - 3 weeks. The strength of bone marrow suppression caused by anthracyclines was higher than that of cyclosporine. Bone marrow suppression was more likely to occur when taxane drugs were used in combination with cyclosporine. Bone marrow suppression could lead to low white blood cells, low blood counts, and even leukemia. Neutropiuria could also be accompanied by infection. 2. ** Intestinal reaction **: - This included loss of appetite, nausea, vomiting, diarrhea, or constipation. Cyclocyclosporine's digestive tract reactions were manifested as loss of appetite, nausea and vomiting, which generally disappeared after stopping the drug for 1 - 3 days. Anthracyclines would cause digestive disorder, such as nausea, vomiting and diarrhea. The digestive tract reactions of taxane drugs were common but not serious, and a few could have diarrhea. 3. ** Cardiac toxicity (mainly due to anthracyclines)**: - The greater the dosage, the greater the cardiac toxicity. There were three types according to the time of appearance: - Acuteness: It often occurs within a few hours or days after administration, and is manifested as cardiac transmission disorder and cardiac arrest. In a few cases, it is manifested as pericarditis and acute left heart failure. - "Chronical: It occurs within 1 year of chemotherapy, and it is manifested as left cardiovascular malfunction, which can eventually lead to heart failure." - Late-onset: It occurs several years after chemotherapy, and may manifest as heart failure, myocardiopathic disease, and cardiac arrest. 4. ** Hair Loss **: - 60 - 90% of patients who took the anthracyclines would have hair loss, which was generally irreversible. After stopping the drug, hair would grow back; mild hair loss was more common with taxane drugs; taxol, a commonly used drug for breast cancer, could also cause hair loss. 5. ** Others **: - Cytoxan has urological reactions. If a large dose of cyclosporine is used and there is no effective preventive measures, it can cause bleeding cystitis, which is manifested as bladder irritation symptoms, oliguria, hematuria, and proteinuria. The occurrence rate is relatively low when the conventional dose is used. There may also be stomatitis, toxic leukemia, skin hyperchrominosis, menstrual disorder, and lung edema. Poisonous Poisonous (appearing on the side of the tongue and sub-tongue on the fifth to tenth day of medication). There were reports of occasional fever, chills, hives, hyperchromism, and joint pain; Allergy to yew drugs (Paclitaxel-like and taxotere can cause it, but not taxol. Pretreatment with hormones and other drugs is required before infusion. The drip rate can be adjusted for mild symptoms. For serious reactions, the drug should be stopped.), peripheral nervous system toxicity (Numbness of fingers and toes is the most common. Obvious sensory and motor disturbances and decreased tendon reflexes may occur at high doses. Grand mal seizure during infusion is reported in some cases.), cardiovascular toxicity (Temporary heart rate and low blood pressure are more common), joint and muscle pain (about half of the patients will feel pain 2 - 3 days after taking the medicine, which is related to the dosage and will recover within a few days. When combined with the White Increasing Needle, the muscle pain will worsen), changes in the liver and gallbladder system (such as elevation of Bilirubin, Alkaline Phosphatase, and Glutamate Oxalic Transaminases in some patients after taking taxol). In addition, chemotherapy drugs could also cause local reactions, such as phlebitis caused by chemotherapy drugs, local skin rupture and necrosis caused by drug exudate, as well as immune suppression, which would cause the body's immune function to be low and the resistance to infection. There were also internal organ damage, such as liver and kidney function damage. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
Do breast cancer drugs have to be taken all the time? How long will it take to be effective?
For breast cancer, this was a long-term treatment. The shortest effective treatment period was five years, and it usually took five to ten years. The main effect of breast cancer hormones was to block the stimulation of estrogens on breast cancer cells, but there was no specific time to see the effect because it was affected by many factors, such as individual differences in patients, cancer stage, severity of the disease, and so on. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
Do breast cancer chemotherapy drugs affect liver function?
The drugs used to treat breast cancer had an effect on liver function. For example, among the commonly used chemotherapy drugs for breast cancer, cyclosporine may cause adverse reactions such as toxic hepatectomy, while anthracyclines may have side effects such as cardiac toxicity, bone marrow suppression, hair loss, myospasm, and gastric and intestinal disturbances. Although the effect on liver function was not explicitly mentioned, the overall toxicity of the drug may indirectly affect liver function. The main toxic reactions of taxane drugs were concentrated in blood toxicity, allergic reactions, peripheral nervous system toxicity, etc. However, multi-drug combination chemotherapy may also affect liver function. In addition, some breast cancer patients showed obvious liver damage after chemotherapy. Their transminase levels rose to more than 1000 (normally, it was only 40). This situation needed to be taken seriously and liver protection and supportive treatment should be carried out in time. It is recommended that the liver function should be checked regularly during the chemotherapy period. If there is damage, the liver protection should be given. <a href="/?from=ask_words" style="color:red" target="_blank">Read more exciting novels for free</a>
What are some inspiring triple negative breast cancer success stories?
Jane is an example. She had triple negative breast cancer. Through early detection and prompt treatment, she was able to fight it off. Her treatment involved a series of chemotherapy sessions. She was brave during the whole process, and now she's been in remission for three years. She often says that early detection was key for her.
Triple Neg Breast Cancer Success Stories: How did they achieve success?
Many achieved success through early detection. Detecting triple negative breast cancer at an early stage allows for more effective treatment. For example, some patients had regular self - breast exams and caught the cancer early. Then they had surgeries like lumpectomy or mastectomy followed by chemotherapy and radiation, which led to their recovery.